I recently have been diagnosed with diabetes (type 2, I believe) but also now have to inject insulin. Will I have to inject insulin for the rest of my life?
You are raising a question that concerns at least one in two individuals worldwide—either that someone in their closest surroundings is affected or they don’t know yet that they too might be at risk of developing diabetes later in life.
Unfortunately, the way we are diagnosed and treated for diabetes mostly happens via high blood sugar (HBS) results. Yet, high blood sugar is a result not the cause of the disease!
Blood sugar imbalances may point to a lack of insulin production, genetic predispositions or of insulin resistance. Simplified, insulin levels determine the main difference between type 1 and type 2 diabetes.
• Type 1 diabetes caused HBS levels can be due to certain genetic predispositions (such as gluten intolerance or full-blown celiac disease) or of hypoinsulinism (low or no insulin production).
• Type 2 diabetes caused HBS levels are mostly due to insulin resistance and much of the time involves hyperinsulinism (excess insulin levels or production).
Clearly, while HBS leads to a mainstream medicine diagnosis of diabetes, not all diabetics are alike nor should their condition be addressed alike.
However, most doctors simply follow the prescription recommendations of the pharmaceutical drug producers. Thus, prescription mostly is based on HBS results—not on insulin production levels.
Many type 2s, therefore, receive extra insulin (“to reduce their HBS levels”) despite their body already over-producing its own insulin and them having turned insulin resistant. No wonder, therefore, that even more unprocessed sugar is deposited in their fatty tissues, adding to weight gain, increased heart and kidney disease risk and, obviously, ever growing insulin resistance.
It surely defies logic to systematically prescribe additional insulin to every type 2 patient— possibly with already high insulin production levels—if they fail to control their blood sugar levels. On the other hand, genetic lack of insulin production seems to intuit the prescription of additional insulin.
Luckily, change in treatment approach is on the horizon. Increasingly, the medical system is starting to acknowledge shortcomings of its approach to diabetes. The focus is shifting from diagnosis and treatment approaches mainly based on HBS levels to one based on insulin production levels.
February 27, 2011 ScienceDaily reported the new availability of a simple home-based and stable urine test that measures the ability to produce insulin in patients with type 1 and type 2 diabetes (source and date of availability in Canada unknown!).
The test was developed at the U.K. Peninsula Medical School by an Exeter-based team lead by Professor Andrew Hattersley and addresses urine C-peptide creatinine ratios. Read more
As a simple home test, this new test replaces multiple blood tests that would have to be taken at a hospital or lab. It is marketed particularly for its advantages to children. Unfortunately, this way of marketing devalues the test’s unique value as an important distinction tool between hypo and hyper insulin production.
Meanwhile, we patients bear the responsibility of pointing out to our GPs and endocrinologists the importance of insulin production level measurements (at different times of the day and over several days) before simply accepting their insulin injection prescriptions.
Only you can make the decision about what will work best for you and your body. Raising relevant questions is important. Have your doctor help you or consult with a qualified natural medicine professional before you decide on your customized plan of action.